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| Trichorrhexis nodosa examined by scanning electron microscopy |
| Guan-Zhao Liang1, Wen-Qi Xu1, Xiao-Li Zheng, Jia Liu, Gui-Xia Lv1, Yong-Nian Shen1, Wei-Da Liu1, Dong-Mei Shi2* |
1Department of Mycology, Hospital for Skin Diseases (Institute of Dermatology), Chinese Academy of Medical Sciences and Peking Union Medical College, Nanjing, Jiangsu 210042, China.
2Department of Dermatology, The Affiliated Hospital of Qingdao University, Qingdao, Shandong 266000, China.
3Department of Dermatology, Jining No.1 People’s Hospital, Jining, Shandong 272011, China. |
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Abstract Introduction
Trichorrhexis nodosa (TN), one of the most common hair shaft anomalies, was first identified by Samuel Wilks in 1852[1]. As either a congenital or acquired hair shaft disorder, TN is characterized by fragile and easily broken hair accompanied by frayed cortical fibers and loss of the cuticle[2]. Complete fracture of the hair shaft subsequently occurs. Moreover, hair shaft fragility may increase the risk of developing TN. Congenital TN, including trichothiodystrophy[3], argininosuccinic aciduria[4], Menkes syndrome[5] and trichohepatoenteric syndrome[6], is rare in the clinical setting. Acquired TN, which is provoked by hair injury secondary to physical or chemical trauma (usually physical trauma), is often overlooked in daily life. We herein reported a case of TN in a woman with androgenetic alopecia.
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Received: 09 October 2017
Published: 15 January 2019
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Corresponding Authors:
Dr. Dong-Mei Shi
E-mail: shidongmei28@163.com
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2018, Vol. 1 
