国际皮肤性病学杂志    0  (): 323-325   ISSN: 2096-5540  CN: 32-1880/R  

泌尿生殖道沙眼衣原体持续性复发性感染的分子机制研究
郑波, 王千秋
中国医学科学院、中国协和医科大学皮肤病研究所, 南京210042
收稿日期 2004-11-22  修回日期 null  网络版发布日期 null
参考文献  [1] Belland RJ, Nelson DE, Virok D,et al. Transcriptome analysis of chlamydial growth during IFN-gamma-mediated persistence and reactivation. Proc Natl Acad Sci U S A, 2003, 100:15971-15976.
[2] Stenner-Liewen F, Liewen H, Zapata JM, et al. CADD, a Chlamydia protein that interacts with death receptors. J Biol Chem, 2002, 277:9633-9636.
[3] Morrison RP. New insights into a persistent problem-chlamydial infections. J Clin Invest, 2003, 111:1647-1649.
[4] Perfettini JL, Darville T, Dautry-Varsat A, et al. Inhibition of apoptosis by gamma interferon in cells and mice infected with Chlamydia muridarum (the mouse pneumonitis strain of Chlamydia trachomatis). Infect Immun, 2002, 70:2559-2565.
[5] Whittington WL, Kent C, Kissinger P, et al. Determinants of persistent and recurrent Chlamydia trachomatis infection in young women:results of a multicenter cohort study. Sex Transm Dis, 2001, 28:117-123.
[6] Morrison RP. Differential sensitivities of Chlamydia trachomatis strains to inhibitory effects of gamma interferon. Infect Immun,2000, 68:6038-6040.
[7] Caldwell HD, Wood H, Crane D, et al. Polymorphisms in Chlamydia trachomatis tryptophan synthase genes differentiate between genital and ocular isolates. J Clin Invest, 2003, 111:1757-1769.
[8] Fehlner-Gardiner C, Roshick C, Carlson JH, et al. Molecular basis defining human Chlamydia trachomatis tissue tropism. A possible role for tryptophan synthase. J Biol Chem, 2002, 277:26893-26903.
[9] Read TD, Brunham RC, Shen C, et al. Genome sequences of Chlamydia trachomatis MoPn and Chlamydia pneumoniae AR39.Nucleic Acids Res, 2000, 28:1397-1406.
[10] Peffettini JL, Darville T, Gachelin G, et al. Effect of Chlamydia trachomatis infection and subsequent tumor necrosis factor alpha secretion on apoptosis in the murine genital tract. Infect Immun,2000, 68:2237-2244.
[11] Jendro MC, Fingerle F, Deutsch T, et al. Chlamydia trachomatisinfected macrophages induce apoptosis of activated T cells by secretion of tumor necrosis factor-alpha in vitro. Med Microbiol Immunol (Berl), 2004, 193:45-52.
[12] Jendro MC, Deutsch T, Korber B,et al. Infection of human monocyte-derived macrophages with Chlamydia trachomatis induces apoptosis of T cells:a potential mechanism for persistent infection.Infect Immun, 2000, 68:6704-6711.
[13] Betsou F, Sueur JM, Orfila J. Serological investigation of Chlamydia trachomatis heat shock protein 10. Infect Immun, 1999, 67:5243.
[14] Dean D, Powers VC. Persistent Chlamydia trachomatis infections resist apoptotic stimuli. Infect Immun, 2001,69:2442-2447.
[15] Gérard HC, Whittum-Hudson JA, Schumacher HR,et al. Differential expression of three Chlamydia trachomatis hsp60-encoding genes in active vs. persistent infections. Microb Pathog, 2004, 36:35-39.
[16] Beere HM, Wolf BB, Cain K,et al. Heat-shock protein 70 inhibits apoptosis by preventing recruitment of procaspase-9 to the Apaf-1apoptosome. Nat Cell Biol, 2000, 2:469-475.
[17] Dong F, Sharma J, Xiao Y,et al. Intramolecular dimerization is required for the chlamydia-secreted protease CPAF to degrade host transcriptional factors. Infect Immun, 2004, 72:3869-3875.
[18] Fan P, Dong F, Huang Y, et al. Chlamydia pneumoniae secretion of a protease-like activity factor for degrading host cell transcription factors required for major histocompatibility complex antigen expression. Infect Immun, 2002, 70:345-349.
[19] Gerard HC, Kohler L, Branigan PJ, et al. Viability and gene expression in Chlamydia trachomatis during persistent infection of cultured human monocytes. Med Microbiol Immunol (Berl),1998, 187(2):115-120.
[20] Perfettini JL, Reed JC, Israel N, et al. Role of Bcl-2 family members in caspase-independent apoptosis during clamydia infection.Infect Immun, 2002, 70:55-61.

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