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国际皮肤性病学杂志 2005 31 (2): 113-115 ISSN: 2096-5540 CN: 32-1880/R |
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朗格汉斯细胞与人类乳头瘤病毒感染 |
缪旭, 骆丹 |
南京医科大学第一附属医院皮肤科 210029 |
收稿日期 2004-03-05 修回日期 null 网络版发布日期 null |
参考文献 [1] Matthews K, Leong CM, Baxter L,et al. Depletion of Langerhans cells in human papillomavirus type 16-infected skin is associated with E6-mediated down regulation of E-cadherin. J Virol, 2003, 77:8378-8385. [2] Giannini SL, Hubert P, Doyen J, et al. Influence of the mucosal epithelium microenvironment on Langerhans cells: implications for the development of squamous intraepithelial lesions of the cervix. Int J Cancer, 2002, 97:654-659. [3] El-Sherif AM, Seth R, Tighe PJ, et al. Decreased synthesis and expression of TGF-beta1, beta2, and beta3 in epithelium of HPV 16-positive cervical precancer: a study by microdissection, quantitative RT-PCR, and immunocytochemistry. J Pathol, 2000, 192:494. [4] al-Saleh W,Giannini SL,Jacobs N, et al.Correlation of T-helper secretory differentiation and types of antigen-presenting cells in squamous intraepithelial lesions of the uterine cervix.J Pathol,1998,184:283-290. [5] Hubert P, van den Brule F, Giannini SL, et al. Colonization of in vitro-formed cervical human papillomavirus-associated (pre)neoplastic lesions with dendritic cells: role of granulocyte/macrophage colony-stimulating factor. Am J Pathol, 1999, 154:775-784. [6] Sanclemente G, Gill DK. Human papillomavirus molecular biology and pathogenesis. J Eur Acad Dermatol Venereol, 2002, 16:231. [7] Memar OM, Arany I, Tyring SK. Skin-associated lymphoid tissue in human immunodeficiency virus-1, human papillomavirus, and herpes simplex virus infections. J Invest Dermatol, 1995, 105(1 Suppl): S99- S104. [8] Sobhani I, Walker F, Aparicio T, et al. Effect of anal epidermoid cancer-related viruses on the dendritic (Langerhans') cells of the human anal mucosa. Clin Cancer Res, 2002, 8:2862-2869. [9] Coleman N, Birley HD, Renton AM, et al. Immunological events in regressing genital warts. Am J Clin Pathol, 1994, 102:768-774. [10] Fausch SC, Da Silva DM, Kast WM. Differential uptake and cross-presentation of human papillomavirus virus-like particles by dendritic cells and Langerhans cells. Cancer Res, 2003, 63:3478-3482. [11] Fausch SC, Da Silva DM, Rudolf MP, et al. Human papillomavirus virus-like particles do not activate Langerhans cells: a possible immune escape mechanism used by human papillomaviruses.J Immunol, 2002, 169:3242-3249. [12] Mota F, Rayment N, Chong S, et al. The antigen-presenting environment in normal and human papillomavirus (HPV)-related premalignant cervical epithelium. Clin Exp Immunol, 1999, 116:33-40. [13] Connor JP, Ferrer K, Kane JP, et al. Evaluation of Langerhans' cells in the cervical epithelium of women with cervical intraepithelial neoplasia. Gynecol Oncol, 1999, 75:130-135. [14] Woodworth CD, Simpson S. Comparative lymphokine secretion by cultured normal human cervical keratinocytes, papillomavirus-immortalized, and carcinoma cell lines. Am J Pathol, 1993, 142:1544. [15] Frazer IH, Thomas R, Zhou J, et al. Potential strategies utilised by papillomavirus to evade host immunity. Immunol Rev, 1999, 168:131-142. [16] Morelli AE, Sananes C, Di Paola G, et al. Relationship between types of human papillomavirus and Langerhans' cells in cervical condyloma and intraepithelial neoplasia. Am J Clin Pathol, 1993, 99:200-206. |
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