国际皮肤性病学杂志    2004 30 (6): 366-368   ISSN: 2096-5540  CN: 32-1880/R  

过敏性接触性皮炎免疫反应机制的研究进展
郭丽芳, 范卫新
南京医科大学第一附属医院皮肤科 210029
收稿日期 2004-01-06  修回日期 null  网络版发布日期 null
参考文献  [1] Banchereau J, Briere F, Caux C, et al. lmmunobiology of dendritic cells. Annu Rev lmmunol, 2000,18:767-811.
[2] Watanabe H, Unger M, Tuvel B, et al. Contact hypersensitivity: the mechanism of immune responses and T cell balance. J Interferon Cytokine Res, 2002, 22:407-412.
[3] Mizumoto N, Iwabichi K, Nakanura H, et al. Enhanced contact hypersensitivity in human monocyte chemoattractant protein-1 transgenic mouse. Immunobiology, 2001, 204:477-493.
[4] Weiss JM, Renkl AC, Maier CS, et al. Osteopontin is involved in the initiation of cutaneous contact hypersensitivity by inducing Langerhans and dendritic cell migration to lymph nodes. J Exp Med, 2001, 194:1219-1229.
[5] Tsushima F, Iwai H, Otsuki N, et al. Preferential contribution of B7-H1 to programmed death-1-mediated regulation of hapten-specific allergic inflammatory responses. Eur J Immunol, 2003, 33:2773.
[6] Shimizu T, Abe R, Nishihira J, et al. Impaired contact hypersensitivity in macrophage nigration inhibitory factor-deficient mice. Eur J Immunol, 2003, 33:1478-1487.
[7] Kabashima K, Murata T, Tanaka H, et al. Thromboxane A2 modulates interaction of dendritic cells and T cells and regulates acquired immunity. Nat Immunol, 2003, 4:694-701.
[8] Grabbe S, Schwarz T. hnmunoregulatory mechanisms involved in elicitation of allergic contact hypersensitivity, Immunol Today, 1998, 19:37-44.
[9] Dimitroff C J, Kupper TS, Sackstein R. Prevention of leukocyte migration to inflamed skin with a novel fluorosugar modifier of cutaneous lymphocyte-associated antigeu. J Clin Invest, 2003, 112:1008.
[10] Piguet PF, Grau GE, Hauser C, et al. Tumor necrosis factor is a critical mediator in hapten induced irritant and contact hypersensitivity reactions. J Exp Med, 1991, 173:673-679.
[11] Nakae S, Naruse-Nakajima C, Sudo K, et al. IL-1 alpha, but not IL-1 beta, is required for contact-allergen-specific T cell activation during the sensitization phase in contact hypersensitivity. Iht Immunol, 2001, 13:1471-1478.
[12] Yawalkar N, Egli F, Brand CU, et al. Antigen-presenting cells and keratinocytes express interleukin-12 in allergic contact dermatitis.Contact Dermatitis, 2000, 42:18-22.
[13] Steinbrink K, Sorg C, Macher E. Low zone tolerance to contact allergens in mice: a functional role for CD8+T helper type 2 cells. J Exp Med, 1996, 183:759-768.
[14] Xu H, DiIulio NA, Fairchild RL. T cell populations primed by hapten sensitization in contact sensitivity are distinguished by polarized patterns of cytokine production: interferon gamma-producing (Tcl) effector CD8+T cells and interleukin (Ⅱ) 4/Ⅱ-10-producing (Th2) negative regulatory CD4+T cells. J Exp Med, 1996, 183:1001-1012.
[15] Salerno A, Dieli F, Sireci G, et al. Interleukin-4 is a critical cytokine in contact sensitivity. Immunology, 1995, 84:404-409.
[16] Maurer M, Seidel-Guyenot W, Metz M, et al. Critical role of IL-10in the induction of low zone tolerance to contact allergens. J Clin Invest, 2003, 112:432-439.
[17] Luger TA, Scbolzen TE, Brzoska T, et al. New insights into the functions of alpha-MSH and related peptides in the immune system. Ann N Y Acad Sci, 2003, 994:133-140.
[18] Xu B, Bulfone-Paus S, Aoyama K, et al. Role of Fas/Fas ligand-mediated apoptosis in murine contact hypersensitivity. Int Immunopharmacol, 2003, 3:927-938.
[19] Kawase Y, Hoshino T, Yokota K, et al. Exacerbated and prolonged allergic and non-allergic inflmnmatory cutaneous reaction in nice with targeted interleukin-18 expression in the skin. J Invest Dermatol,2003,121:502-509.
[20] Plitz T, Saint-Mezard P, Satho M, et al. IL-18 binding protein protects against contact hypersensitivity. J Immunol, 2003, 171:1164.
[21] Xu H, Banerjee A, Dilulio NA, et al. Development of effector CD8 +T cells in contact hypersensitivity occurs independently of CD4+T cells. J Immunol, 1997, 158:4721-4728.
[22] Bouloe A, Cavani A, Katz SI. Contact hypersensitivity in MHC class Il-deficient mice depends on CD8 T lymphocytes primed by immunostimulating Langerhans cells. J Invest Dermatol, 1998, 111:44-49.
[23] Bour H, Peyron E, Gaucherand M, et al. Major histocompatibility complex class I-restricted CD8+T cells and class I-restricted CD4 +T cells, respectively, mediate and regulale contact sensitivity to dinitrofluorobenzene. Eur J Immunol, 1995, 25:3006-3010.
[24] Dearman RJ, Kimber I. Role of CD4(+) T helper 2-type cells in cutaneous inflammatory responses induced by fluorescein isothiocyanate. Immunology, 2000, 101:442-451.
[25] DiIulio NA, Xu H, Fairchild RL. Diversion of CD4+T cell development from regulatory T helper to effector T helper cells alters the contact hypersensitivity response. Eur J Immunol, 1996, 26:2606-2612.

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