国际皮肤性病学杂志    2004 30 (2): 102-104   ISSN: 2096-5540  CN: 32-1880/R   活化诱导的细胞死亡与皮肤病 田艳丽, 王刚 第四军医大学西京医院全军皮肤性病中心, 西安 710032 收稿日期 2003-05-12  修回日期 null  网络版发布日期 null 参考文献  [1] De Panfilis G.‘Activation-induced cell death':a special program able to preserve the homeostasis of the skin? Exp Dermatol, 2002, 11(1):1-11. [2] Hussein MR, Haemel AK, Wood GS. Apoptosis and melanoma:molecular mechanisms. J Pathol, 2003, 199(3):275-288. [3] Maldonado MA, MacDonald GC, Kakkanaiah VN, et al. Differential control of autoantibodies and lymphoproliferation by Fas ligand expression on CD4+ and CD8+ T cells in vivo. J Immunol, 1999, 163(6):3138-3142. [4] van der Burg M, de Groot R, Comans-Bitter WM, et al. Autoimmune lymphoproliferative syndrome(ALPS)in a child from consanguineous parents:a dominant or recessive disease? Pediatr Res, 2000, 47(3):336-343. [5] De Panfilis G, Caruso A, Sansoni P, et al. Identification of Fas-L-expressing apoptotic T lymphocytes in normal human peripheral blood:in vivo suicide. Am J Pathol, 2001,158(2):387-391. [6] Tanaka M, Itai T, Adachi M, et al. Downregulation of Fas ligand by shedding. Nat Med, 1998, 4(1):31-36. [7] Su X, Cheng J, Liu W, et al. Autocrine and paracrine apoptosis are mediated by differential regulation of Fas ligand activity in two distinct Jurkat T cell populations. J Immunol, 1998, 160(11):5288-5293. [8] Musci MA, Latinis KM, Koretzky GA. Signaling events in T lymphocytes leading to cellular activation or programmed cell death. Clin Immunol Immunopathol, 1997, 83(3):205-222. [9] Hodge MR, Ranger AM, Charles de la Brousse F, et al. Hyperproliferation and dysregulation of IL-4 expression in NF-ATp-deficient mice. Immunity, 1996, 4(4):397-405. [10] Boise LH, Minn AJ, Noel PJ, et al. CD28 costimulation can promote T cell survival by enhancing the expression of Bcl-XL. Immunity,1995, 3(1):87-98. [11] Friesen C, Herr I, Krammer PH, et al. Involvement of the CD95(APO-1/FAS)receptor/ligand system in drug-induced apoptosis in leukemia cells. Nat Med, 1996, 2(5):574-577. [12] Ozawa M, Ferenczi K, Kikuchi T,et al. 312-nanometer ultraviolet B light(narrow-band UVB)induces apoptosis of T cells within psoriatic lesions. J Exp Med, 1999, 189(4):711-718. [13] Lonati A, Licenziati S, Canaris AD, et al. Reduced production of both Th1 and Tcl lymphocyte subsets in atopic dermatitis(AD). Clin Exp Immunol, 1999, 115(1):1-5. [14] Gilhar A, Shalaginov R, Assy B, et al. Alopecia areata is a T-lymphocyte mediated autoimmune disease:lesional human T-lymphocytes transfer alopecia areata to human skin grafts on SCID mice. J Investig Dermatol Syrup Proc, 1999,4(3):207-210. [15] Schnyder B, Frutig K, Mauri-Hellweg D,et al. T-cell-mediated cytotoxicity against keratinocytes in sulfamethoxazol-induced skin reaction.Clin Exp Allergy, 1998, 28(11):1412-1417. [16] Henseleit U, Zhang J, Wanner R, et al. Role of p53 in UVB-induced apoptosis in human HaCaT keratinocytes. J Invest Dermatol, 1997,109(6):722-727. [17] Leverkus M, Yaar M, Gilchrest BA. Fas/Fas ligand interaction contributes to UV-induced apoptosis in human keratinocytes. Exp Cell Res, 1997, 232(2):255-262. [18] Kasibhatla S, Brunner T, Genestier L, et al. DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1. Mol Cell, 1998, 1(4):543-551. [19] Gutierrez-Steil C, Wrone-Smith T, Sun X, et al. Sunlight-induced basal cell carcinoma tumor cells and ultraviolet-B-irradiated psoriatic plaques express Fas ligand(CD95L). J Clin Invest, 1998, 101(1):33-39. [20] Buechner SA, Wernli M, Harr T, et al. Regression of basal cell carcinoma by intralesional interferon-alpha treatment is mediated by CD95(Apo-1/Fas)-CD95 ligand-induced suicide. J Clin Invest,1997, 100(11):2691-2696. [21] Aragane Y, Maeda A, Cui CY, et al. Inhibition of growth of melanoma cells by CD95(Fas/APO-1)gene transfer in vivo. J Invest Dermatol, 2000, 115(6):1008-1014. [22] Zaks TZ, Chappell DB, Rosenberg SA, et al. Fas-mediated suicide of tumor-reactive T cells following activation by specific tumor:selective rescue by caspase inhibition. J Immunol, 1999, 162(6):3273-3279. 通讯作者: