国际皮肤性病学杂志    2002 28 (2): 99-102   ISSN: 2096-5540  CN: 32-1880/R   毛囊免疫赦免与斑秃 赵玉磊, 范卫新, 汤爱民 南京医科大学第一附属医院皮肤科, 210029 收稿日期 2001-11-06  修回日期 null  网络版发布日期 null 参考文献  [1] Shapiro J,Madani S.Alopecia areata:diagnosis and management.Int J Dermatol,1999,38 (Suppl 1):19-24. [2] Madani S,Shapiro J.Alopecia areata update.J Am Acad Dermatol,2000,42(4):549-566. [3] Pans R,Slominski A,Czametzki BM.Is alopecia areata an autoimmune-response against melanogenesis-related proteins,exposed by abnormal MHC class I expression in the anagen hair bulb? Yale J Biol Med,1993,66(6):541-554. [4] Pans R,Christoph T,Muller-Rover S.Immunology of the hair follicle:a short journey into term incognita.J Investig Dermatol Syrup Proc,1999,4(3):226-234. [5] Streilein JW.Unraveling immune privilege.Science,1995,270(5239):1158-1159. [6] Ferguson TA,Griffith TS.A vision of cell death:insights into immune privilege.Immunol Rev,1997,156:167-184. [7] Billingham RE,Silvers WK.A biologist's reflections on dermatology.J Invest Dermatol,1971,57(4):227-240. [8] Westgate GE,Craggs RI,Gibson WT.Immune privilege in hair grewth.J Invest Dennatol,1991,97(3):417420. [9] Christoph T,Muller-Rover S,Audring H,et al.The human hair follicle immune systern:cellular compositian and immune privilege.Br J Dermatol,2000,142(5):862-873. [10] Moresi JM,Horn TD.Distribution of Langerhans cells in human hair follicle.J Cutan Pathol,1997,24(10):636-640. [11] Griffith TS,Bnmner T,Fletcher SM,et al.Fas ligand-induced apeptesis as a mechanism of immune privilege.Science,1995,270 (5239):1189-1192. [12] Chen JJ,Sun Y,Nabel GJ.Regulation of the proinflammatory effects of Fas ligand (CD95L).Science,1998,282(5394):1714-1717. [13] Sabelko KA,Kelly KA,Nahm MH,e al.Fas and Fas ligand ènhance the pathogenesis of experimental allergic encephalomyelitis,but are not essential for immune privilege in the central nervous system.J Immunol,1997,159(7):3096-3099. [14] McElwee KJ,Tobin DJ,Bystryn JC,et al.Alopecia areata:an autoim-mune disease? Exp Dermatol,1999,8(5):371-379. [15] Zhang JG,Oliver RF.Immunohistological study of the development of the cellular infiltrate in the pelage follicles of the DEBR model for alopecia areata.Br J Dermatol,1994,130(4):405-414. [16] McElwee KJ,Spiers EM,Oliver RF.In vivo depletion of CD8+ T cells restores hair growth in the DEBR model for alopecia areata.Br J Dermatol,1996,135(2):211-217. [17] Freyschmidt-Paul P,Seiter S,ZoUer M,et al.Treatment with an anti-CD44v10-specific antibody inhibits the onset of alopecia areata in C3H/HeJ mice.J Invest I)ermatol,2000,115(4):653-657. [18] Gilhar A,Ullmann Y,Berkutzki T,et al.Autoimmune hair loss (alopecia areata) transferred by T lymphocytes to human scalp explants on SCID mice.J Clin Invest,1998,101(1):62-67. [19] Gilhar A,Sbalaginov R,Assy B,et al.Alopecia areata is a T-lymphocyte mediated antoimmune disease:lesianal human T-lymphocytes transfer alopecia areata to human skin grafts on SaD mice.J Investig Dermatol Symp Proc,1999,4(3):207-210. [20] Bodemer C,Peuchmaar M,Fraitaig S,et al.Role of cytoto:dc T cells in chronic alopecia areata.J Invest Dermatol,2000,114(1):112-116. 通讯作者: