国际皮肤性病学杂志    2002 28 (1): 20-22   ISSN: 2096-5540  CN: 32-1880/R   红茶与皮肤疾病关系的研究进展 张素慧1, 吕俊华1, 张宏2 1. 暨南大学医学院药理学教研室 广州 510632; 2. 暨南大学华侨医院皮肤科 收稿日期 2001-05-16  修回日期 null  网络版发布日期 null 参考文献  [1] Yang CS, Wang ZY. Tea and cancer. J Natl Cancer Inst, 1993,85(13):1038-1049. [2] Gremwrod C, Campjbell D. Tea and health: a research program. Tea & Coffee Trade J, 1996,168(6): 32. [3] International Agency for Research of Cancer. Coffee, tea, mate, methylxanthines and methylglyoxal. IARC Monogr Eval Carcinog Risks HUM, 1991,51:1-513. [4] Graham HN. Green tea composition, consumption, and polyphenol chemistry. Prey Med, 1992, 21(3): 334-350. [5] Lu YP, Lou YR, Xie JG, et al. Inhibitory effect of black tea on the growth of established skin tumors in mice: effects on tumor size, apoptosis, mitosis and bromodeoxyuridine incorporation into DNA. Carcinogenesis, 1997,18(11): 2163-2169. [6] Javed S, Mehrotra NK, Shukla Y. Chemopreventive effects of black tea polyphenols in mouse skin model of carcinogenesis. Biomed Fnviron Sci,1998, 11(4): 307-313. [7] Miller DL, Weinstock MA. Nonmelanoma skin cancer in the United States: incidence. J Am Acad Dermatol,1994, 30(5 Pt 1):774-778. [8] Huang MT, Xie JG, Wang ZY, et al. Effects of tea, decaffeinated tea,and caffeine on UVB light-induced complete carcinogenesis in SKH-1mice: demonstration of caffeine as a biologically important constituent of tea. Cancer Res, 1997, 57(13): 2623-2629. [9] Record IR, Drecsti IE. Protection by tea against UV-A + B-induced skin cancers in hairless mice. Nutr Cancer, 1998,32(2): 71-75. [10] Wang ZY, Huang MT, Lou YR, et al. Inhibitory effects of black tea,green tea, decaffeinated black tea, and deceffeinated green tea on ultraviolet B light-induced skin carcinogenesis in 7, 12-dimethylbenz [ a ] anthracene-initiated SKH-1 mice. Cancer Res, 1994, 54(13): 3428-3435. [11] Katiyar SK, Mulhtar H. Inhibition of phorbol ester tumor promoter 12-O-tetradecanoylphorbol-13-acetate-caused intlammatory responses in SENCAR mouse skin by black tea polyphenols. Carcinogenesis, 1997,18(10):1911-1916. [12] Record IR, Dreosti IE. Protection by black tea and green tea against UVB and UVA + B induced skin cancer in hairless mice.Mutat Res, 1998,422(1): 191-199. [13] Zhao J, Jin X, Yaping E, et al, Photoprotective effect of black tea extracts against UVB-induced phototoxicity in skin. Photochem Photobiol, 1999, 70(4): 637-644. [14] Tedesco AC, Martinez L, Gonzalez S. Photochemistry and photobiology of actinic erythema: defensive and reparative cutaneous mechanisms. Braz J Med Biol Res, 1997, 30(5): 561-575. [15] Weisburger JH, Rivenson A, Kingston DG, et al. Dietary modulation of the carcinogenicity of the heterocyclic amines. Princess Takamatsu Symp,1995,23: 240-250. [16] Parshad R, Sanford KK, Price FM, et al. Protective action of plant polyphenols on radiation-induced chromatid breaks in cultured human cells. Anticancer Res, 1998, 18(5A): 3263-3266. [17] Wei H, Zhang X, Zhao JF, et al. Scavenging of hydrogen peroxide and inhibition of ultraviolet light-induced oxidative DNA damage by aqueous extracts from green and black teas. Free Radic Biol Med, 1999, 26(11 ～12): 1427-1435. [18] Ahmed NU, Ueda M, Nikaido O, et al. High levels of 8-hydroxy-2'-deoxyguanosine appear in normal human epidemmis after a single dose of ultraviolet radiation. Br J Dermatol, 1999,140(2): 226-231. [19] Hattori Y, Nishigori C, Tanaka T, et al. 8-hydroxy-2'-deoxyguanosine is increased in epidermal cell of hairless mice after chronic ultraviolet B exposure. J Invest Dermatol, 1997, 107(2): 733-737. 通讯作者: