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1. �㽭��ҽѧԺ���������е�������ҽԺƤ����, ����310009; | |||||||||||||||||||||||||||||||||||||||||||||||||||
1. �㽭��ҽѧԺ���������е�������ҽԺƤ����, ����310009; | |||||||||||||||||||||||||||||||||||||||||||||||||||
Abstract: | |||||||||||||||||||||||||||||||||||||||||||||||||||
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[1] Lee S J, Benveniste EN. Adhesion molecule expression and regulation on cells of the central nervous system. J Neuroimmunol, 1999, 98:77. [2] Neitmann M, Alexander M, Brinckmann J, et al. Attachment and chemotaxis of melanocytes after ultraviolet irradiation in vitro. Br J Dermatol, 1999, 141:794-801. [3] Jouneau A, Yu YQ, Pasdar M, et al. Plasticity of cadherin-catenin expression in the melanocyte lineage. Pigment Cell Res, 2000, 13:260-272. [4] Danen EH, Jansen KF, Klein CE, et al. Loss of adhesion to basement membrane components but not to keratinocytes in proliferating melanocytes. Eur J Cell Biol, 1996, 70:69-75. [5] Takano N, Kawakami T, Kawa Y,et al. Fibronectin combined with stem cell factor plays an important role in melanocyte proliferation,differentiation and migration in cultured mouse neural crest cells. Pigment Cell Res, 2002,15:192-200. [6] Boissy RE. Melanosome transfer to and translocation in the keratinocyte. Exp Dermatol, 2003,12 ( Suppl 2 ):5-12. [7] Ameen M, Exarchou V, Chu AC. Topical calcipotriol as monotherapy and in combination with psoralen plus ultraviolet A in the treatment of vitiligo. Br J Dermatol, 2001, 145:476-479. [8] Hedley SJ, Metcalfe R, Gawkrodger DJ,et al. Vitiligo melanocytes in long-term culture show normal constitutive and cytokine-induced expression of intercellular adhesion molecule-1 and major histocompatibility complex class �� and class �� molecules. Br J Dermatol, 1998,139:965-973. [9] Hwang JH, Ahn JS, Kim SD, et al. The changes of serum soluble intercellular adhesion molecule-1 after systemic steroid treatment in vitiligo. J Dermatol Sci, 1999,22:11-16. [10] Abdallah M, Abdel-Naser MB, Moussa MH, et al. Sequential immunohistochemical study of depigmenting and repigmenting minigrafts in vitiligo. Eur J Dermatol, 2003, 13:548-552. [11] Gauthier Y, Cario-Andre M, Lepreux S, et al. Melanocyte detachment after skin friction in non lesional skin of patients with generalized vitiligo. Br J Dermatol, 2003,148:95-101. [12] Le Poole IC, van den Wijngaard RM, Westerhof W, et al. Tenascin is overexpressed in vitiligo lesional skin and inhibits melanocyte adhesion. Br J Dermatol, 1997,137:171-178. [13] Jimbow K, Chen H, Park JS, et al. Increased sensitivity of melanocytes to oxidative stress and abnormal expression of tyrosinase-related protein in vitiligo. Br J Dermatol, 2001,144:55-65. [14] Mou KH, Zhang XQ, Yu B, et al. Promoting of melanocyte adhesion and migration by Malytea Scurfpea fruit in vitro. Methods Find Exp Clin Pharmacol, 2004, 26:167-170. [15] Lu Y, Zhu WY, Tan C, et al. Melanocytes are potential immunocompetent cells: evidence from recognition of immunological characteristics of cultured human melanocytes. Pigment Cell Res, 2002, 15:454-460. [16] Iuga AO, Qureshi AA, Lerner EA. Nitric oxide is toxic to melanocytes in vitro. Pigment Cell Res, 2004, 17:302-306. [17] Jamal S, Schneider RJ. UV-induction of keratinocyte endothelin-1downregulates E-cadherin in melanocytes and melanoma cells. J Clin Invest, 2002, 110:443-452. [18] Morandini R, Boeynaems JM, Hedley SJ, et al. Modulation of ICAM-1 expression by alpha-MSH in human melanoma cells and melanocytes. J Cell Physiol, 1998, 175:276-282. [19] Gauthier Y, Cario Andre M, Taieb A. A critical appraisal of vitiligo etiologic theories. Is melanocyte loss a melanocytorrhagy? Pigment Cell Res, 2003, 16:322-332. [20] Nishimura EK, Jordan SA, Oshima H, et al. Dominant role of the niche in melanocyte stem-cell fate determination. Nature, 2002, 416:854-860. |
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