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[2] Moore MM, Rifas-Shiman SL, Rich-Edwards JW, et al. Perinatal predictors of atopic dermatitis occurring in the first six months of life.Pediatrics, 2004, 113:468-474.
[3] Leung DY, Boguniewicz M, Howell MD, et al. New insights into atopic dermatitis. J Clin Invest, 2004, 113:651-657.
[4] Novak N, Bieber T, Leung DY. Immune mechanisms leading to atopic dermatitis. J Allergy Clin Immunol, 2003, 112(6 Suppl):S128-S139.
[5] Lee YA, Wahn U, Kehrt R, et al. A major susceptibility locus for atopic dermatitis maps to chromosome 3q21. Nat Genet, 2000, 26:470-473.
[6] Cookson WO, Ubhi B, Lawrence R, et al. Genetic linkage of childhood atopic dermatitis to psoriasis susceptibility loci. Nat Genet,2001, 27:372-373.
[7] Beyer K, Nickel R, Freidhoff L, et al. Association and linkage of atopic dermatitis with chromosome 13q12-14 and 5q31-33 markers. J Invest Dermatol, 2000,115:906-908.
[8] Soderhall C, Bradley M, Kockum I, et al. Linkage and association to candidate regions in Swedish atopic dermatitis families. Hum Genet,2001, 109:129-135.
[9] Bradley M, Soderhall C, Luthman H, et al. Susceptibility loci for atopic dermatitis on chromosomes 3, 13, 15, 17 and 18 in a Swedish population. Hum Mol Genet, 2002, 11:1539-1548.
[10] Donnadieu E, Jouvin MH, Rana S, et al. Competing functions encoded in the allergy-associated F ( c ) epsilonRIbeta gene. Immunity,2003, 18:665-674.
[11] Tsunemi Y, Saeki H, Nakamura K, et al. Interleukin-13 gene polymorphism G4257A is associated with atopic dermatitis in Japanese patients. J Dermatol Sci, 2002, 30:100-107.
[12] He JQ, Chan-Yeung M, Becker AB, et al. Genetic variants of the IL13 and IL4 genes and atopic diseases in at-risk children. Genes Immun, 2003, 4:385-389.
[13] Walley AJ, Chavanas S, Moffatt MF, et al. Gene polymorphism in Netherton and common atopic disease. Nat Genet, 2001, 29:175-178.
[14] Kabesch M, Carr D, Weiland SK, et al. Association between polymorphisms in serine protease inhibitor, kazal type 5 and asthma phenotypes in a large German population sample. Clin Exp Allergy,2004, 34:340-345.
[15] Kaburagi Y, Shimada Y, Nagaoka T, et al. Enhanced production of CC-chemokines (RANTES, MCP-1, MIP-1alpha, MIP-1beta,and eotaxin) in patients with atopic dermatitis. Arch Dermatol Res,2001, 293:350-355.
[16] Kabesch M, Peters W, Carr D, et al. Association between polymorphisms in caspase recruitment domain containing protein 15 and allergy in two German populations. J Allergy Clin Immunol, 2003, 111:813-817.
[17] Ahmad-Nejad P, Mrabet-Dahbi S, Breuer K,et al. The toll-like receptor 2R753Q polymorphism defines a subgroup of patients with atopic dermatitis having severe phenotype. J Allergy Clin Immunol,2004, 113:565-567.
[18] Benn CS, Melbye M, Wohlfahrt J, et al. Cohort study of sibling effect, infectious diseases, and risk of atopic dermatitis during first 18months of life. BMJ, 2004, 328:1223.
[19] Gustafsson D, Sjoberg O, Foucard T. Sensitization to food and airborne allergens in children with atopic dermatitis followed up to 7years of age. Pediatr Allergy Immunol, 2003, 14:448-452.
[20] Sator PG, Schmidt JB, Honigsmann H. Comparison of epidermal hydration and skin surface lipids in healthy individuals and in patients with atopic dermatitis. J Am Acad Dermatol, 2003, 48:352-358.
[21] Arakawa S, Hatano Y, Katagiri K. Differential expression of mRNA for Th1 and Th2 cytokine-associated transcription factors and suppressors of cytokine signalling in peripheral blood mononuclear cells of patients with atopic dermatitis. Clin Exp Immunol, 2004, 135: 505-510.
[22] Akdis M, Trautmann A, Klunker S, et al. T helper (Th) 2 predominance in atopic diseases is due to preferential apoptosis of circulating memory/effector Th1 cells. FASEB J, 2003, 17:1026-1035.
[23] Nomura I, Gao B, Boguniewicz M, et al. Distinct patterns of gene expression in the skin lesions of atopic dermatitis and psoriasis: a gene microarray analysis. J Allergy Clin Immunol, 2003, 112:1195.