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Abstract:

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DOI:
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[2] Oppel T, Schuller E, Gunther S, et al. Phenotyping of epidermal dendritic cells allows the differentiation between extrinsic and intrinsic forms of atopic dermatitis. Br J Dermatol, 2000, 143:1193-1198.
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[9] Novak N, Kruse S, Kraft S, et al. Dichotomic nature of atopic dermatitis reflected by combined analysis of monocyte immunophenotyping and single nucleotide polymorphisms of the interleukin-4/interleukin-13 receptor gene: the dichotomy of extrinsic and intrinsic atopic dermatitis. J Invest Dermatol, 2002, 119:870-875.
[10] Li XM, Kleiner G, Huang CK, et al. Murine model of atopic dermatitis associated with food hypersensitivity. J Allergy Clin Immunol, 2001, 107:693-702.
[11] Sampson HA. The evaluation and management of food allergy in atopic dermatitis. Clin Dermatol, 2003, 21:183-192.
[12] Sampson HA. Utility of food-specific IgE concentrations in predicting symptomatic food allergy. J Allergy Clin Immunol, 2001, 107:891-896.
[13] Higaki S, Morohashi M, Yamagishi T, et al. Comparative study of staphylococci from the skin of atopic dermatitis patients and from healthy subjects. Int J Dermatol, 1999, 38:265-269.
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[16] Matsushima H, Hayashi S, Shimada S. Skin scratching switches immune responses from Th2 to Th1 type in epicutaneously immunized mice. J Dermatol Sci, 2003, 32:223-230.
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