[an error occurred while processing this directive] | ����Ƥ���Բ�ѧ��־ 2005, 31(2) 81-83 DOI: ISSN: 2096-5540 CN: 32-1880/R | ||||||||||||||||||||||||||||||||||||||||||||||||||
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Abstract: | |||||||||||||||||||||||||||||||||||||||||||||||||||
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�ո����� 2004-05-08 ������ ����淢������ | |||||||||||||||||||||||||||||||||||||||||||||||||||
DOI: | |||||||||||||||||||||||||||||||||||||||||||||||||||
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[1] Hussein AM. Chemotherapy-induced alopecia: new developments. South Med J,1993, 86:489-496. [2] Hidalgo M, Rinaldi D, Medina G,et al. A phase I trial of topical topitriol (calcitriol, 1,25-dihydroxyvitamin D3) to prevent chemotherapy-induced alopecia. Anticancer Drugs, 1999, 10:393-395. [3] Hussein AM, Jimenez JJ, McCall CA, et al. Protection from chemotherapy-induced alopecia in a rat model. Science, 1990, 249:1564-1566. [4] Paus R, Handjiski B, Eichmuller S, et al. Chemotherapy-induced alopecia in mice. Induction by cyclophosphamide, inhibition by cyclosporine A, and modulation by dexamethasone. Am J Pathol, 1994, 144:719-734. [5] Slominski A, Paus R, Plonka P, et al. Pharmacological disruption of hair follicle pigmentation by cyclophosphamide as a model for studying the melanocyte response to and recovery from cytotoxic drug damage in situ. J Invest Dermatol, 1996, 106:1203-1211. [6] Lindner G, Botchkarev VA, Botchkareva NV, et al. Analysis of apoptosis during hair follicle regression (catagen).Am J Pathol, 1997, 151:1601-1617. [7] Tobin DJ, Hagen E, Botchkarev VA, et al. Do hair bulb melanocytes undergo apoptosis during hair follicle regression (catagen)? J Invest Dermatol, 1998, 111:941-947. [8] Muller-Rover S, Rossiter H, Paus R, et al. Overexpression of Bcl-2 protects from ultraviolet B-induced apoptosis but promotes hair follicle regression and chemotherapy-induced alopecia. Am J Pathol, 2000, 156:1395-1405. [9] Davis ST, Benson BG, Bramson HN, et al. Prevention of chemotherapy-induced alopecia in rats by CDK inhibitors. Science, 2001, 291:134-137. [10] Lakin ND, Jackson SP. Regulation of p53 in response to DNA damage. Oncogene, 1999, 18:7644-7655. [11] Botchkarev VA, Komarova EA, Siebenhaar F, et al. p53 Involvement in the control of murine hair follicle regression. Am J Pathol, 2001, 158:1913-1919. [12] Botchkarev VA. Molecular mechanisms of chemotherapy-induced hair loss. J Investig Dermatol Symp Proc, 2003, 8:72-75. [13] Botchkarev VA, Komarova EA, Siebenhaar F, et al. p53 is essential for chemotherapy-induced hair loss. Cancer Res, 2000, 60:5002-5006. [14] Sharov AA, Li GZ, Palkina TN, et al. Fas and c-kit are involved in the control of hair follicle melanocyte apoptosis and migration in chemotherapy-induced hair loss. J Invest Dermatol, 2003, 120:27-35. [15] Ekholm SV, Reed SI. Regulation of G(1) cyclin-dependent kinases in the mammalian cell cycle. Curr Opin Cell Biol, 2000, 12:676-684. [16] Song S, Lambert PF. Different responses of epidermal and hair follicular cells to radiation correlate with distinct patterns of p53 and p21 induction. Am J Pathol, 1999, 155:1121-1127. [17] Paus R, Schilli MB, Handjiski B,et al. Topical calcitriol enhances normal hair regrowth but does not prevent chemotherapy-induced alopecia in mice. Cancer Res, 1996, 56:4438-4443. [18] Schilli MB, Paus R, Menrad A. Reduction of intrafollicular apoptosis in chemotherapy-induced alopecia by topical calcitriol-analogs. J Invest Dermatol, 1998, 111:598-604. [19] Hussein AM, Stuart A, Peters WP. Protection against chemotherapy-induced alopecia by cyclosporin A in the newborn rat animal model. Dermatology, 1995, 190:192-196. [20] Maurer M, Handjiski B, Paus R. Hair growth modulation by topical immunophilin ligands: induction of anagen, inhibition of massive catagen development, and relative protection from chemotherapy-induced alopecia. Am J Pathol, 1997 , 150:1433-1441. [21] Danilenko DM, Ring BD, Yanagihara D, et al. Keratinocyte growth factor is an important endogenous mediator of hair follicle growth, development, and differentiation. Normalization of the nu/nu follicular differentiation defect and amelioration of chemotherapy-induced alopecia. Am J Pathol, 1995, 147:145-154. [22] Peters EM, Foitzik K, Paus R, et al. A new strategy for modulating chemotherapy-induced alopecia, using PTH/PTHrP receptor agonist and antagonist. J Invest Dermatol, 2001, 117:173-178. [23] Sato N, Leopold PL, Crystal RG. Effect of adenovirus-mediated expression of Sonic hedgehog gene on hair regrowth in mice with chemotherapy-induced alopecia. J Natl Cancer Inst, 2001, 93:1858-1864. [24] Komarov PG, Komarova EA, Kondratov RV, et al. A chemical inhibitor of p53 that protects mice from the side effects of cancer therapy. Science, 1999, 285:1733-1737. [25] Ohnemus U, Unalan M, Handjiski B,et al. Topical estrogen accelerates hair regrowth in mice after chemotherapy-induced alopecia by favoring the dystrophic catagen response pathway to damage. J Invest Dermatol, 2004, 122:7-13. |
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