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HPV������ֲ����Ƥ�������о���չ
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1. ��ž�����һһҽԺƤ���Բ���, �Ϻ� 200081;
2. �ڶ���ҽ��ѧ����ҽԺƤ����;
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1. ��ž�����һһҽԺƤ���Բ���, �Ϻ� 200081;
2. �ڶ���ҽ��ѧ����ҽԺƤ����;
3. ��ž�����һһҽԺ������
1. ��ž�����һһҽԺƤ���Բ���, �Ϻ� 200081;
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[2] Parant E, Jumbou O, Dantal J, et al. Viruses involved in allograft recipients'cutaneous carcinomas. Ann Dermatol Venereol, 2001, 128(8-9):871-875.
[3] Antonsson A, Forslund O, Ekberg H, et al. The ubiquity and impressive genomic diversity of human skin papillomaviruses suggest a commensalic nature of these viruses. J Virol, 2000, 74(24):11636-11641.
[4] Boxman IL, Berkhout RJ, Mulder LH, et al. Detection of human papillomavirus DNA in plucked hairs from renal transplant recipients and healthy volunteers. J Invest Dermatol, 1997, 108(5):712-715.
[5] Euvrard S, Kanitakis J, Claudy A. Cutaneous complications after organ transplant. Presse Med, 1999, 28(33):1833-1838.
[6] de Jong-Tieben LM, Berkhout R J, ter Schegget J, et al. The prevalence of human papillomavirus DNA in benign keratotic skin lesions of renal transplant recipients with and without a history of skin cancer is equally high:a clinical study to assess risk factors for keratotic skin lesions and skin cancer. Transplantation, 2000, 69(1):44-49.
[7] de Villiers EM, Lavergne D, McLaren K, et al. Prevailing papillomavirus types in non-melanoma carcinomas of the skin in renal allograft recipients. Int J Cancer, 1997, 73(3):356-361.
[8] Berkhout R J, Bouwes Bavinck JN, ter Schegget J. Persistence of human papillomavirus DNA in benign and(pre)malignant skin lesions from renal transplant recipients. J Clin Microbiol, 2000, 38(6):20872096.
[9] Mansat-Krzyzanowska E, Dantal J, Hourmant M, et al. Frequency of mucosal HPV DNA detection(types 6/11, 16/18, 31/35/51)in skin lesions of renal transplant patients. Transpl Int, 1997, 10(2):137-140.
[10] Arends MJ, Benton EC, McLaren KM,et al. Renal allograft recipients with high susceptibility to cutaneous malignancy have an increased prevalence of human papillomavirus DNA in skin tumours and a greater risk of anogenital malignancy. Br J Cancer, 1997, 75(5):722-728.
[11] Ozsaran AA, Ates T, Dikmen Y, et al. Evaluation of the risk of cervical intraepithelial neoplasia and human papilloma virus infection in renal transplant patients receiving immunosuppressive therapy. Eur J Gynaecol Oncol, 1999, 20(2):127-130.
[12] Brown MR, Noffsinger A, First MR, et al. HPV subtype analysis in lower genital tract neoplasms of female renal transplant recipients. Gynecol Oncol, 2000, 79(2):220-224.
[13] Furumoto H, Irahara M. Human papilloma virus(HPV)and cervical cancer. J Med Invest, 2002, 49(3-4):124-133.
[14] zur Hausen H. Immortalization of human cells and their malignant conversion by high risk human papillomavirus genotypes. Semin Cancer Biol, 1999, 9(6):405-41l.
[15] Filippova M, Song H, Connolly JL,et al. The human papillomavirus
[16] E6 protein binds to tumor necrosis factor(TNF)R1 and protects cells from TNF-induced apoptosis. J Biol Chem, 2002, 277(24):21730-21739.16.Gibson GE, OGrady A, Kay EW, et al. Langerhans cells in benign,premalignant and malignant skin lesions of renal transplant recipients and the effect of retinoid therapy. J Eur Acad Dermatol Venereol,1998, 10(2):130-136.
[17] Bouwes Bavinck JN, Claus FH, Hardie DR, et al. Relation between HLA antigens and skin cancer in renal transplant recipients in Queensland, Australia. J Invest Dermatol, 1997, 108(5):708-711.
[18] Barba A, Tessari G, Boschiero L, et al. Renal transplantation and skin diseases:review of the literature and results of a 5-year followup of 285 patients. Nephron, 1996, 73(2):131-136.