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��Ƥ��Բ�ѧ��־ 2002, 28(6) 356-359 DOI: ISSN: 2096-5540 CN: 32-1880/R

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[1] Jimenez-Cervantes C, Martinez-Esparza M, Perez C, et al. Inhibition of melanogenesis in response to oxidative stress:transient downregulation of melanocyte differentiation markers and possible involvement of microphthalmia transcription factor. J Cell Sci, 2001, 114 (Pt 12):2335-2344.
[2] Schallreuter KU, Moore J, Wood JM, et al. Epidermal H (2) O (2) accumulation alters tetrahydrobiopterin (6BH₄) recycling in vitiligo:identification of a general mechanism in regulation of all 6BH₄-dependent processes? J Invest Dermatol, 2001, 116 (1):167-174.
[3] Schallreuter KU, Wood JM, Pittelkow MR, et al. Increased monoamine oxidase A activity in the epidermis of patients with vitiligo. Arch Dermatol Res, 1996, 288(1):14-18.
[4] Schallreuter KU. A review of recent advances on the regulation of pigmentation in the human epidermis. Cell Mol Biol (Noisy-legrand), 1999, 45(7):943-949.
[5] Schallreuter KU, Wood JM, Pittelkow MR, et al. Regulation of melanin biosynthesis in the human epidermis by tetrahydrobiopterin. Science, 1994, 263(5152):1444-1446.
[6] Schallreuter KU, Buttner G, Pittelkow MR, et al. Cytotoxicity of 6-biopterin to human melanocytes. Biochem Biophys Res Commun,1994, 204(1):43-48.
[7] Kalko SG, Gelpi JL, Fita I, et al. Theoretical study of the mechanisms of substrate recognition by catalase. J Am Chem Soc, 2001, 123(39):9665-9672.
[8] Schallreuter KU, Wood JM, Berger J. Low catalase levels in the epidermis of patients with vitiligo. J Invest Dermatol, 1991,97(6):1081-1085.
[9] Beazley WD, Gaze D, Panske A, et al. Serum selenium levels and blood glutathione peroxidase activities in vitiligo. Br J Dermatol,1999, 141(2):301-303.
[10] Maresca V, Roccella M, Roccella F, et al. Increased sensitivity to peroxidative agents as a possible pathogenic factor of melanocyte damage in vitiligo. J Invest Dermatol, 1997, 109(3):310-313.
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[12] Bartosik J, Wulf HC, Kobayasi T. Melanin and melanosome complexes in long standing stable vitiligo--an ultrastructural study. Eur J Dermatol, 1998, 8(2):95-97.
[13] Yohn JJ, Norris DA, Yrastorza DG, et al. Disparate antioxidant enzyme activities in cultured human cutaneous fibroblasts, keratinocytes,and melanocytes. J Invest Dermatol, 1991, 97 (3):405-409.
[14] Haycock JW, Rowe SJ, Cartledge S, et al. Alpha-melanocytestimulating hormone reduces impact of proinflammatory cytokine and peroxide-generated oxidative stress on keratinocyte and melanoma cell lines. J Biol Chem, 2000, 275(21):15629-15636.
[15] Passi S, Grandinetti M, Maggio F, et al. Epidermal oxidative stress in vitiligo. Pigment Cell Res, 1998, 11 (2):81-85.
[16] Schallreuter KU, Wood JM, Lemke KR, et al. Treatment of vitiligo with a topical application of pseudocatalase and calcium in combination with short-term UVB exposure:a case study on 33 patients. Dermatology, 1995, 190 (3):223-229.
[17] Schallreuter KU. Successful treatment of oxidative stress in vitiligo.Skin Pharmacol Appl Skin Physiol, 1999, 12 (3):132-138.
[18] Schallreuter KU, Moore J, Wood JM, et al. In vivo and in vitro evidence for hydrogen peroxide (H₂O₂) accumulation in the epidermis of patients with vitiligo and its successful removal by a UVB-activated pseudocatalase. J Investig Dermatol Symp Proc, 1999, 4 (1):91-96.

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